If, in the well-fed state, glycolysis increases until the liver's glucose-to-glycogen stores are filled, we want to create more glycogen. If NADH inhibits the citric acid cycle, while NAD⁺ and ADP activate the citric acid cycle, why do high levels of NADH and FADH₂ increase the rate of the electron transport chain (ETC)? Why is there during the well-fed state high levels of

What is the ratio of NADH and FADH₂ to NAD⁺ and FAD in the well-fed state versus the fasting state?

With excess glucose stores, ETC shuts down, NADH and FADH2 buildup. I don't understand this, what am I understanding wrong

With depleted energy, NAD+ and FAD+ are high